Characteristics of self-regulation of the epidemic process of infection caused by the Epstein–Barr virus (Herpesviridae: <i>Lymphocryptovirus</i>, HHV-4)

Introduction. Among the available scientific literature, there are no publications addressing processes of self-regulation in parasite-host population systems with reference to chronic infections, including infection caused by EpsteinBarr virus (EBV infection).
 The aim study is assess manifestations epidemic process EBV through lens basic tenets theory parasitic systems.
 Materials and methods. was performed using data from selected such database sources as Scopus, Web Science, Cochrane Library, PubMed, CyberLeninka, RSCI, etc. list analyzed included published articles authors this study, reporting results retrospective epidemiological analysis incidence infectious mononucleosis Russia general Moscow particular, well laboratory tests regarding detection frequency specific antibodies proteins.
 Results. course promotes a close long-term interaction between pathogen host. genetic variability functions nonspecific human immune defense play key role two heterogeneous populations underlie their phasal self-transformation. A variety social natural factors (adverse chemical, physical, biological, climatic impacts, etc.) trigger reactivation infection, thus providing continuous existence additional host population.
 Conclusion. context understanding underlying unevenness its process. obtained can be adjusted for other infections having similar transmission mechanisms life cycles (including herpes infections) map out strategies control spread aerosol pathogen.